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As IRS-1 and IRS-2 are thought to mediate most of the effects of IR and IGF-1R in breast cancer cells, it may be possible to disrupt this molecule without affecting normal glucose homeostasis mediated by other adapter proteins in insulin target organs

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Rather, it reflects the evidence available, how the compound is manufactured and distributed, and whether regulators believe it should be available through established medical channels

Vimal J, George NA, Kumar RR, Kattoor J, Kannan S

Functionalization of Nanomaterials for Skin Cancer Theranostics